当前位置: X-MOL 学术Nat. Rev. Endocrinol. › 论文详情
Our official English website, www.x-mol.net, welcomes your feedback! (Note: you will need to create a separate account there.)
Pancreatic δ-cells influence the glycaemic set point
Nature Reviews Endocrinology ( IF 40.5 ) Pub Date : 2024-02-01 , DOI: 10.1038/s41574-024-00956-2
Olivia Tysoe

Somatostatin, produced by pancreatic δ-cells, has been known to inhibit insulin secretion since its discovery in the 1970s. However, the role of somatostatin in the regulation of the glycaemic set point (the homeostatic blood level of glucose between meals) remains unclear. A study in Nature Metabolism now explores the contribution of pancreatic δ-cell somatostatin secretion to the glycaemic set point and β-cell insulin production.



中文翻译:

胰腺 δ 细胞影响血糖设定点

生长抑素是由胰腺 δ 细胞产生的,自 20 世纪 70 年代发现以来,已知它可以抑制胰岛素分泌。然而,生长抑素在血糖设定点(两餐之间的血糖稳态血液水平)调节中的作用仍不清楚。《自然代谢》杂志上的一项研究现在探讨了胰腺 δ 细胞生长抑素分泌对血糖设定点和 β 细胞胰岛素产生的影响。

更新日期:2024-02-02
down
wechat
bug